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TY - JOUR TI - Iodine Deficiency Disorders in Indonesia: Past, Present, and Future PY - 2023/06/04 Y2 - 2025/11/09 JF - TEMU ILMIAH NASIONAL PERSAGI JA - TINPERSAGI VL - 1 SE - 2. PLENO TIN PERSAGI TAHUN 2005 UR - https://tin.persagi.org/index.php/tin/article/view/128 SP - 75-99 AB - Iodine Deficiency (ID) may cause a wide spectrum of abnormalities affecting fetus, neonates, children and adult population. The most severe form is endemic cretinism which is found in severely IDD areas. The pathophysiology of these are the inavailability of thyroid hormone during neurophysical development beginning in early pregnancy which are irreversible. In moderate and mild ID more subtle anomalies may be found, ranging from unsuccessful pregnancy outcomes (abortion, perinatal death congenital abnormalities etc). Hypothyroxinemia not hypo-T3 is crucial. for brain development, since delicate regulation of T3 can be done by local production of T3 via 5'-Dll deiodinase. Preterm babies are prone to hypothyroxinemia and inadequate iodine intake and this should be prevented. The highest burden of iodine deficiency is not goitre but psychoneurophysical development of men, which mostly are irreversible neurological damage. Th!s problem is easily prevented by continuous supply of adequate iodine supply to high risk populations (pregnant and nursing mothers, children esp underthree's and child bearing age women). The recent International Thyroid Congress in Buenos Aires recently (Oct 31, 2005 _ attached in this paper) issued a statement that all its members should help their government to overcome this global problem. ER -